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Autoimmune neurological conditions associated with zika virus infection

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dc.creatorAcosta Ampudia, Yeny Yasbleidyspa
dc.creatorMonsalve Carmona, Diana Marcelaspa
dc.creatorCastillo-Medina, Luis F.spa
dc.creatorRodríguez Velandia, Yhojan Alexisspa
dc.creatorPacheco Nieva, Yovanaspa
dc.creatorHalstead, Susanspa
dc.creatorWillison, Hugh J.spa
dc.creatorAnaya, Juan-Manuelspa
dc.creatorRamírez Santana, Heily Carolinaspa
dc.creator.googleAcosta-Ampudia, Yeny
dc.creator.googleMonsalve, Diana M.
dc.creator.googleCastillo-Medina, Luis F.
dc.creator.googleRodríguez, Yhojan
dc.creator.googlePacheco, Yovana
dc.creator.googleHalstead, Susan
dc.creator.googleWillison, Hugh J.
dc.creator.googleAnaya, Juan-Manuel
dc.creator.googleRamirez-Santana, Carolina
dc.date.accessioned2019-09-27T13:22:09Z
dc.date.available2019-09-27T13:22:09Z
dc.date.created2018spa
dc.date.issued2018
dc.description.abstractZika virus (ZIKV) is an emerging flavivirus rapidly spreading throughout the tropical Americas. Aedes mosquitoes is the principal way of transmission of the virus to humans. ZIKV can be spread by transplacental, perinatal, and body fluids. ZIKV infection is often asymptomatic and those with symptoms present minor illness after 3 to 12 days of incubation, characterized by a mild and self-limiting disease with low-grade fever, conjunctivitis, widespread pruritic maculopapular rash, arthralgia and myalgia. ZIKV has been linked to a number of central and peripheral nervous system injuries such as Guillain-Barré syndrome (GBS), transverse myelitis (TM), meningoencephalitis, ophthalmological manifestations, and other neurological complications. Nevertheless, mechanisms of host-pathogen neuro-immune interactions remain incompletely elucidated. This review provides a critical discussion about the possible mechanisms underlying the development of autoimmune neurological conditions associated with Zika virus infection. © 2018 Acosta-Ampudia, Monsalve, Castillo-Medina, Rodríguez, Pacheco, Halstead, Willison, Anaya and Ramírez-Santana.eng
dc.format.mimetypeapplication/pdf
dc.identifier.doi10.3389/fnmol.2018.00116
dc.identifier.issn1662-5099
dc.identifier.urihttps://repository.urosario.edu.co/handle/10336/20351
dc.language.isoengspa
dc.relation.citationTitleFrontiers in Molecular Neuroscience
dc.relation.citationVolumeVol. 11
dc.relation.ispartofFrontiers in Molecular Neuroscience, ISSN:1662-5099, Vol. 11 (2018)spa
dc.relation.urihttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC5904274/pdf/fnmol-11-00116.pdfspa
dc.rights.accesRightsinfo:eu-repo/semantics/openAccess
dc.rights.accesoAbierto (Texto completo)spa
dc.source.bibliographicCitationAhmad, S.S.Y., Amin, T.N., Ustianowski, A., Zika virus: Management of infection and risk (2016) BMJ, 352spa
dc.source.instnameinstname:Universidad del Rosario
dc.source.reponamereponame:Repositorio Institucional EdocUR
dc.subjectAedesspa
dc.subjectArthralgiaspa
dc.subjectAutoimmunityspa
dc.subjectBody Fluidspa
dc.subjectConjunctivitisspa
dc.subjectGuillain Barre Syndromespa
dc.subjectMaculopapular Rashspa
dc.subjectMeningoencephalitisspa
dc.subjectMolecular Mimicryspa
dc.subjectMyelitisspa
dc.subjectNeuropathologyspa
dc.subjectNonhumanspa
dc.subjectReviewspa
dc.subjectVirus Infectionspa
dc.subjectVirus Transmissionspa
dc.subjectZika Virusspa
dc.subject.ddcEnfermedades
dc.subject.lembEnfermedades autoinmunesspa
dc.subject.lembAutoinmunidadspa
dc.subject.lembFluído corporalspa
dc.titleAutoimmune neurological conditions associated with zika virus infectionspa
dc.typerevieweng
dc.type.hasVersioninfo:eu-repo/semantics/publishedVersion
dc.type.spaRevisiónspa
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