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In Utero Fetal Programming and Its Impact on Health in Adulthood

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dc.creatorRamírez-Vélez, Robinson
dc.date.accessioned2020-05-26T00:06:45Z
dc.date.available2020-05-26T00:06:45Z
dc.date.created2012spa
dc.description.abstractAdverse events during intrauterine life may program organ growth and favor disease later in life. This is the usually called 'Barker's hypothesis'. Increasing evidence suggests that conditions like vascular disease, hypertension, metabolic syndrome, and type 2 diabetes mellitus are programmed during the early stages of fetal development and become manifest in late stages of life, when there is an added impact of lifestyle and other conventional acquired environmental risk factors that interact with genetic factors. The aim of this review was to provide additional, updated evidence to support the association between intrauterine fetal health and increased prevalence of chronic non-communicable diseases in adulthood. Various potential cellular and molecular mechanisms proposed to be related to the above hypothesis are discussed, including endothelial function, oxidative stress, insulin resistance, and mitochondrial function. © 2011 SEEN.eng
dc.format.mimetypeapplication/pdf
dc.identifier.doihttps://doi.org/10.1016/j.endonu.2012.02.002
dc.identifier.urihttps://repository.urosario.edu.co/handle/10336/23925
dc.language.isoengspa
dc.relation.citationEndPage393
dc.relation.citationIssueNo. 6
dc.relation.citationStartPage383
dc.relation.citationTitleEndocrinologia y Nutricion
dc.relation.citationVolumeVol. 59
dc.relation.ispartofEndocrinologia y Nutricion, Vol.59, No.6 (2012); pp. 383-393spa
dc.relation.urihttps://www.scopus.com/inward/record.uri?eid=2-s2.0-84863469050&doi=10.1016%2fj.endonu.2012.02.002&partnerID=40&md5=79b57045d4291b4673f1dd3136a50916spa
dc.rights.accesRightsinfo:eu-repo/semantics/openAccess
dc.rights.accesoAbierto (Texto Completo)spa
dc.source.instnameinstname:Universidad del Rosariospa
dc.source.reponamereponame:Repositorio Institucional EdocURspa
dc.subject.keywordAdulthoodspa
dc.subject.keywordCell functionspa
dc.subject.keywordDiseasesspa
dc.subject.keywordEndothelium cellspa
dc.subject.keywordFetus developmentspa
dc.subject.keywordHealth statusspa
dc.subject.keywordHumanspa
dc.subject.keywordInsulin resistancespa
dc.subject.keywordMitochondrionspa
dc.subject.keywordNon communicable diseasespa
dc.subject.keywordOxidative stressspa
dc.subject.keywordPrevalencespa
dc.subject.keywordShort surveyspa
dc.subject.keywordAdultspa
dc.subject.keywordBirth weightspa
dc.subject.keywordBlood vesselsspa
dc.subject.keywordChronic diseasespa
dc.subject.keywordFetal developmentspa
dc.subject.keywordHumansspa
dc.subject.keywordInsulin resistancespa
dc.subject.keywordMetabolismspa
dc.subject.keywordMitochondriaspa
dc.subject.keywordCoronary heart diseasespa
dc.subject.keywordFetal growth retardationspa
dc.subject.keywordInsulin resistancespa
dc.subject.keywordMaternal nutritionspa
dc.titleIn Utero Fetal Programming and Its Impact on Health in Adulthoodspa
dc.title.TranslatedTitleProgramación Fetal in utero y su impacto en la salud del adultospa
dc.typearticleeng
dc.type.hasVersioninfo:eu-repo/semantics/publishedVersion
dc.type.spaArtículospa
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